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Veterinary
Orthopedic Manipulation and (Early Detection, Treatment and
Prevention) By
Wm. L. Inman BS, DVM, CVCP Introduction As we all love our canine friends, we reserve
a particular fondness for the larger of the dog breeds. They seem to be more
interesting and portend more personality and vitality than their smaller
cousins. It is particularly common for veterinary
clients to announce that they are not "small dog people" but rather
prefer the larger or giant breeds. "They are ‘real’ dogs"
as they will say. The giant breeds also carry with them a very
specific set of disease conditions. In some cases these conditions can be
life-threatening and at the very least can be financially and emotionally
devastating. I will never forget the sight of one of my
best clients, (a tall, stately man, always impeccably dressed, in a corporate
power-suit and always looking in control). He was standing outside the doorway
of my veterinary practice, covered in blood and vomitus, with the dead carcass
of his beloved Great Dane, "Goliath" dangling in his arms. Whenever I want to remember what significant
personal loss is like, I always flash back in my mind to the expression of total
helplessness and loss on the face of this man. Goliath was a 6-year old male brindle Great
Dane who 3 hours before was the love of this man’s life and the pride of his
family. Goliath was very thirsty and after a big drink of water decided to
engage in a good tug-of-war with his brother in the back yard. The gastric
dilatation-torsion that occurred took Goliath’s life within two hours. By
the time Goliath was noted as not feeling well and rushed to my hospital, he was
terminal. Goliath died in the owner’s lap in the car on the way to my
hospital. Two months previously the same man and his
family had presented the 9-month-old "brother" of the gastric torsion
dog with painful limping in the right front leg. This was a black or sable Dane,
and upon x-ray evaluation he was found to have defects in the bone at the top
end of his humerus. He had Osteochondritis Dissicans (OCD) of the
shoulder. Immediately, the other shoulder and the rear legs were
radiographed to make sure that "Buster" was not just limping on the
leg that hurt the most. Buster was radiographically clear, so we opted
to operate on the shoulder and remove the joint defect. The operation was a
success. Within 6 weeks he was back terrorizing Goliath with incessant
games of tug-of-war and wrestling matches. I can’t help but think that if I
hadn’t done such a good job with Buster’s surgery that he wouldn’t have
been active enough to roughhouse with Goliath on a full stomach. This tragic
event may have been averted. That hindsight did not change the fact that
Goliath was now presented dead outside my hospital door. His 130# held up by his
owner, wearing a ruined business suit and surrounded by his family. With a belly
the size of a basketball and his tongue blue, Goliath had gone through the
agonal throws of death in the car, while stuck in Seattle traffic and the whole
family helplessly watching. The patriarch of this family was a captain of
industry and a multi-millionaire in the computer industry. He had a beautiful
family and every blessing known to man. I believe he would have given them all
up if I could have just helped Goliath. I pronounced his dog dead and
this stately man broke down and began to cry like a child. It was he who had
given the dog a large bowl of water and then turned both Danes out in the back
yard to play. Six months after Goliath died, he presented me
with Buster who now was having trouble with the other shoulder.
Radiographs revealed that he had developed OCD in the other shoulder and in the
stifle and would need surgery in both places. At this point, my client had
decided that enough was enough and that this breed was just too fraught with
potential diseases to place any more emotional confidence. The cost of surgery
was not an issue for this man. The emotional expense was too high, and against
my recommendation he opted for euthanasia. That is not the end.
Three years later the eldest son added a black and white Great Dane puppy to his
family against the advice of his father. At 18-months "Marmaduke"
or "Duke" presented unable to stand in the rears. He had poor
control of his front legs. Duke had experienced no trauma, fall or accident. Further examination would reveal that Duke was
a Canine Wobbler (caudal cervical instability) and even though the
problem was in the base of the neck, it affected his rear legs and was
progressing to his front legs. Regardless of medical therapy, Duke became a
quadriplegic within 4 hours and the inevitable euthanasia
would leave this family with 3 dead Great Danes in as many years. Five years later I ran into father and when I
asked how he and his family were doing, he offered that he had not gotten
another dog since the death of his Danes. "Can’t afford ‘em"
was his rationalization. "Those inbred dogs have too many problems.
We’ve sworn of all dogs in our family". How depressing. Discussion Is the Great Dane genetically cursed? Have we
inadvertently bred diseases into the giant breeds by line breeding and even
outcross breeding? What is the cause of these three disease conditions? Could
they all be the same thing and if they are could they be treated effectively?
Better yet, would there be a way to detect these disease states before they
actually produce a clinical problem? Is there an underlying secret that has
escaped the veterinary community for the last 100 years? The Secret
It is not going to endear me to many of my
colleagues to report that a huge medical phenomenon has been missed in the
veterinary field. What has been found is that a neurological
"interference" is present in all these disease conditions.
This interference is caused by the traumas of normal life and manifests itself
as most of the disease conditions we eventually see in our dogs. This neurological phenomenon is called a "subluxation" and is recognized by over 100,000 chiropractors as the cause of disease. It is what chiropractor treat; it is what they reduce when they make an "adjustment". Unfortunately veterinarians have used the term
subluxation for years, but incorrectly. We vets think that if a joint is out of
place, it is said to be "luxated". If it looks like the
bones are less than completely out of joint, it must then be "sub-luxated".
A subluxation is in the nervous tissue and not in the bone. A subluxation cannot
be seen on an x-ray, yet on occasion, its effects can be seen on an x-ray as a
partial dislocation. This is where the two terms get mixed up. My profession labors under the
misconception that a subluxation is anatomical instead of neurological.
This explains why the two professions, veterinary and chiropractic, have not
been able to get together. Chiropractors know that they are able to adjust the
subluxations out of their pets and make them sound. Vets are looking at the
x-rays and failing to see the "anatomical sub-luxations". They cannot
figure out what the chiropractor is treating and so relegates the whole area to
quackery. At no point in any US veterinary teaching
institution’s education curriculum is there any instruction on chiropractic
care, neither theory, nor application.
This is a travesty in that 80% of all animals have neuronal subluxations and
most of those will eventually direct some form of disease condition. A New Approach The practitioners that embrace the possibility
that subluxation can and does cause disease, has opened up a huge universe of
here-to-fore untreatable diseases. He or she becomes a very busy veterinarian
and continues to perform "miracles" with the VOM Technology,
They soon find that this is a means of treating impossible cases and diagnosing
most all of the others. The vet soon realizes that these methods of
rooting out the cause of the disease condition is possible not only after the
dog has a problem, but in most cases can be done months to years before a
problem is seen. Here we have an invaluable treatment tool, but
the diagnostic potentials are unlimited. Finally, the preventative
value for finding subluxation before disease is seen and repairing
them before the disease is expressed, is the crux of this article. It is also
the incredible value of Veterinary Orthopedic Manipulation to dogs in general. VOM began in 1982 and was developed by this
author into a complete diagnostic, treatment and preventative specialty for the
animal health care provider. Based on over 45,000 clinical cases (on
file) the VOM Technology has trained in excess of 4200 practitioners in the
US, Great Britain Sweden, Canada, Australia and New Zealand. It is the
fastest growing veterinary specialty of any kind in the world and yet it is
still relatively unknown to both pet owners and veterinarians alike. It has been
called "the best-kept secret in veterinary medicine". The Diseases What conditions can the VOM Technology
diagnose and treat? How does that involve the giant breeds and the Great Dane
particularly? In the Dane we see these conditions that can
be diagnosed and treated:
Many of these diseases are events that can be the beginning of the end for our dogs, and anything that could prevent them would be a Godsend. VOM can do that. How
is VOM Performed? The
VOM Diagnostic Technology is done with the use of a stainless steel spinal
accelerometer, a sort of spinal hammer that provides a very tiny motion in a
very short period of time. In all the history of the VOM Technology, there has
never been an animal injured with the application of this device. This is also
true of over 6 million applications on the human. The device cannot
create an injury or a subluxation. If
the diagnostic pulse imparts a force to a spinal segment that has a neuronal
interference associate with it, the pet elicits a pathological reflex unique to
that subluxation. One
can evaluate the whole of the pet’s spine and in so doing a
"blueprint" of the patterns of subluxations are seen. These
patterns correspond to disease states, and are exactly reproducible among
animals with identical diseases. These
reflexes are not subtle but are easily seen by the practitioner and the pet
owner, taking all of the guesswork out of the diagnostic process. Previous
chiropractic diagnostic techniques were particularly nebulous and subjective.
VOM makes the diagnostic process completely objective and simple. Virtually,
a child could do it. Once
a diagnosis is reached, the treatment technology is applied. That is done with
the same instrument and the presence of the diagnostic "reads" or
pathological reflexes are negated one by one. On occasion, the reading patterns
change and do not disappear for several more adjustments. This is due to the
complex and chronic nature of how the subluxations get there in the first place.
Suffice to say, the reads are reduced, the subluxations are gone, the animal now
has full neuronal capability and the pet is able to complete healing on its own. How
Many Adjustments Are Needed? A
common concern of clients who consider this care is that if the animal is
treated in this fashion, will they have to continually return for more
adjustments? The answer is no. The average amount of adjustments per case is
5.75 or essentially six adjustments over a four-month cycle of healing. The
good news is that after this cycle is done, like in the case of a broken bone
healing back stronger than before, the area that has been healed does not have
problems again. This imparts a cure as apposed to a temporary fix
that previous medical and surgical treatments provide. How
does a subluxation work? Think
of it as a compact disc that becomes severely scratched and at some point in the
music it is only able to send gibberish to the receiver. The music tract becomes
locked in an endless loop that it cannot escape. It will continue this loop
until something acts on the system to free it or until the equipment fails. The scratch
is the subluxation caused by some injury. The gibberish is the pain
impulses sent to the spinal cord by the peripheral nerves. The receiver
that will misinterpret and amplify that gibberish is a segment of the spinal
cord. The equipment that will eventually fail is the body. Note that the
brain is not really involved. How
does the device reverse the subluxation? The
body doesn’t give us anything we do not need. Unfortunately, in the case of
the subluxation phenomenon, it doesn’t know when to stop, and a subluxation
will continue until the animal dies or an outside force acts upon it. The
injured area of the spine that has a subluxation is "frozen" or
"fixed" for future repair and this is the real reason the body
has this mechanism. When
a cat wakes up, it always stretches. This is an example of subluxations being
reduced automatically and the feline is fortunate to be able to do so. Cats
unfortunately have plenty of subluxations that they cannot spontaneously reduce. As
the neuronal impulses of pain hold in place the subluxation as described above,
the mechano-receptive impulses (motion and force) will reverse the
subluxation at the cellular level of the spinal segment. In short, a mechanical
force directed at the involved spinal segment is all that is needed to reverse
the subluxation phenomenon. This is what all types of musculo-skeletal
manipulation, VOM, Acupuncture, Acupressure, and many other modalities have in
common and why they all work! Why
not use your hands like a chiropractor? It is
too slow. The body can resist motion on the spinal reflex level
down to 20 milliseconds. The fastest that a person can move a joint is 80
milliseconds, which is four times too slow. The device operates at 2-4
milliseconds which is 5-10 times faster than need be, and the animal gets the
subluxation reduced whether he wants to or not, each time, every time. Manual
application of force can be applied to reduce subluxation but the patient
has to be very relaxed and cooperative. Canine
Wobbler’s Syndrome Let’s
look at caudal cervical instability from the standpoint of current veterinary
medical disease application. The
pet has had an injury in or around the cervical vertebrae. This has caused a
subluxation and is compromising the muscles that surround the spine, pulling
them out of alignment (very commonly not detectable on radiographs) and
the ensuing pressure on the spinal cord is causing acute pain and loss of
function to the rear legs. This dog is in big trouble. Initial
presentation to a veterinary clinic will find this dog getting large doses of
corticosteroid and pain medication to try to slow down the swelling of the
spinal cord that is accelerating the problem. Once
stabilized, and after myelography where a radio-opaque dye is injected into the
spinal canal, a decision to operate and stabilize the vertebrae that are
impinging on the nerves is made. The surgery is quite extensive and tricky. Only
the finest of veterinary surgeons will attempt this procedure. The unstable
vertebrae will be fused together using plates, screws and surgical bone glue. When
the big dog wakes up, he wakes up screaming. The surgery is very painful post
operatively. Often the surgeon will pre-medicate the pet with pain medication to
avoid this situation. Before
the surgery the veterinarian makes sure that the owner realizes that even if the
surgery was a supreme success, in 8-10 months the exact same condition
could recur in the vertebral segments in front and behind the
fusion. In this scenario, the surgery has saved the dog’s life but
only postpones the problem for 8-10 months. With
this type of reoccurrence rate, the decision is often made to spare the giant
breed from this temporary fix and the client opts for euthanasia. Thus, a
diagnosis of K-9 Wobbler’s Disease becomes the kiss of death,
particularly in the giant breeds. The
dog that goes down acutely is a candidate for the surgery. This is a means to
save his life. The dog that can walk into the veterinary hospital, (in the
experience of this author), is not only a VOM case, but now the prognosis is
excellent. Not
only are we able to quickly treat these cases, we can get them back into their
normal activities and keep them from reoccurring. The reality is that we
actually induce the system to repair itself completely as opposed to creating an
artificial and temporary "fix". Prognosis
of this disease is excellent if found early. That is the key. If we wait
until the dog becomes paralyzed we will have lost the war. Early diagnosis and
treatment is imperative. Radiographic changes are rarely apparent in the
obvious cases, much less the cases that are not showing clinical signs. The
good news is that subclinical cases will show obvious VOM reads as far ahead as
weeks to years before the disease precipitates. Of course this give rise to an
early warning system and also allows us to treat the disease before it comes to
pass. The giant breeds, particularly the Great Dane, are always screened
in a VOM Practice to uncover a potential disaster waiting to happen.
This usually takes seconds within the body of the examination and is one of the
very best things we can do for the 5-month and older Dane puppy. Canine
Hip Dysplasia Syndrome Go to
a shopping mall and do this. Walk up to 100 strangers and ask them what they
think is the most common veterinary disease that they know. Their answer will be
hip dysplasia 95 out of 100 times. Now ask them how Hip Dysplasia in the dog is
treated and you will be astounded. Their response 87% of the time will be
euthanasia! Yes,
Hip Dysplasia is the most common disease condition known in my profession by the
general public. It is thought to be a disease that is treated by putting the dog
to sleep. If a pet owner is asked what the justification for euthanasia would
be, he responds that K-9 Hip Dysplasia is a genetic disease. A dog from a
sire or dam with the disease is so painful that the only humane thing to do is
to put the animal out of its misery. But
wait, we know of dogs that have K-9 Hip Dysplasia Syndrome and have parents that
are OFA Dysplasia free. We also know that animals that are
"stress-fed" as pups have a greater tendency to the syndrome. Male
dogs that rapidly develop are also more prone to develop the disease as opposed
to the smaller, less physically aggressive females. Vitamin C (and the collagen
growth that it directs) also seems to have an effect on the progression of the
disease. Hip
Dysplasia is categorized as a polygenetic, autosomal recessive trait with
incomplete penetrance. Which means that essentially it occurs or doesn’t,
when it wants to or doesn’t anytime or never. Very confusing. Hip
Dysplasia does not act like a genetic disease at all. As we have selected OFA
negative parents in the breeding cycles, it would take only five generations to
show a significant decrease in the incidence of the disease. In the last 20
years of breeding dogs in this selective fashion, the incidence of the K-9 Hip
Dysplasia Syndrome has not decreased at all, but in most studies has increased
in incidence. Hip
Dysplasia acts like an acquired disease. Some pups in susceptible litters
develop the disease and some do not. The males that are more aggressive are more
prone than the females and well-fed litters that are given "hot" diets
are also more prone. When
you see these pups at 5-12 months, they will seem normal but when evaluated with
the VOM Diagnostic Technology, some of the pups will show reading patterns at T-8-9-10
in the middle of the back. The reflexive response is obvious to the practitioner
and the client. These pups will continue to develop laxity of the hip joints
that will lead to K-9 Hip Dysplasia Syndrome if not treated with VOM. VOM
treated pups will be treated at one week, two weeks after that, three weeks
after that, and be done with pathological reads (and the disease) four weeks
after that. These
treated pups will then "dodge the bullet" of Hip Dysplasia by having
early diagnosis and VOM Treatment. This would then prove that this condition was
an acquired disease held in place by subluxation. The genetic component of the
disease is due to the genetic predisposition to subluxation and
thereby the disease. An
early diagnosis and treatment regimen becomes simple when the cause of Dysplasia
Syndrome is subluxation. The VOM Diagnostic and Treatment Technology is used to
isolate these young dogs at risk, and treat them before they suffer irreparable
changes in their hips. Without
this approach we are forced to wait until it is too late and surgery and other
techniques are just salvage operations… or euthanasia. Gastric
Dilatation-Torsion Complex Gastric
Dilatation-Torsion of Volvulous, as it is called, is the potential disaster that
most Dane owners dread. People who have owned Danes have sited this condition as
one that they grew weary of being careful of. Their
complaint was that they could never let their Dane alone around an unlimited
source of water as they might gorge themselves, twist their stomachs, bloat and
die. This
acts as a self-fulfilling prophecy in that the "carefully watered"
Dane becomes eager to gorge when given the opportunity. It has been a reason
that pet owners have opted not to get another Great Dane. We
are led to believe that we have bred a line of dogs that cannot freely drink
water when they become dehydrated. This is diametrically apposed to survival and
makes no sense. What is happening here? The
stomach, esophagus and pylorus all are controlled by nerves that increase or
decrease their function. In normal digestive operation they maintain a certain
tone and function level. In
some animals these organs have compromised function and hang loose in the
abdomen. Food and water stagnate in the stomach, as it is not being moved
through the gastro-intestinal tract normally. The pendulous stomach then has the
opportunity to flip over and twist off the esophagus and the pylorus. The
disastrous bloat that results is life threatening. Why
do some of these dogs have this problem and some do not? For years we thought it
was an inbred genetic faux-paus. We
know now that when we have a dog with a mega-esophagus that we have subluxation
at T-5-6 and when reduce them with VOM the pet is cleared of the
condition the next day. In
Volvulous, we see the same situation. Reduction of the subluxations at T-6-7-8
will restore normal functionality and tone to the stomach, esophagus and pylorus
and prevent this disease. We
also use this technique after we have to untwist the stomach in a Dane that has
bloated. No amount of VOM will untwist a torsed stomach and surgical
stabilization may be required to hold the organ in place. Untwisting
the gut can save the life of the dog. Surgical stabilization can in most
instances prevent another torsion, but the subluxation that caused the condition
is still in place and needs to be handled. It is
seldom after a torsion is reduced and the subluxations are treated, that
surgical stabilization will be necessary. The good
news is that these big dogs have subluxations that can be reduced months
to years before they bloat. The bad news is that these subluxation
sites are ones that involve the autonomic nervous system and do not give rise to
the classical reflexive reads that are so easily seen in musculo-skeletal
disease. With
this in mind, a routine check-up of one of these "dogs at risk" would
involve a VOM Pass in the T-6-7-8 area and if a subluxation is present it
will be effectively reduced. If not present, no harm will come of the
adjustment. An important point of the VOM Technology is that it can and will only
reduce subluxations and can only turn a switch that is "off" to
"on". In 18
years of application the device and the application of VOM has never harmed a
pet. It just does not produce enough motion to do so. Osteochondritis
Dissicans (OCD) OCD,
as it is called in veterinary medicine, is a bone development problem that
occurs in the shoulder of the young rapidly growing dog. The Dane is a classic
case. At 7-14 months the dog presents favoring one of the front legs, or on
occasion the rear leg. It is often seen on both sides but the dog chooses to
favor the one that hurts the most. Veterinarians
usually diagnose this condition by using a specific x-ray positioning technique
that can show the divot of bone at the end of the humerus or femur that has
refused to undergo normal ossification. Bone
first appears as cartilage and then as the young dog ages, is converted to bone. In
the case of the giant breeds, we have increased weight and stresses unique to
these breeds due to their size. This added to their incredible growth rate,
coupled with metabolic "encouragement" by using diets and supplements
designed to enhance and accelerate grow, you have the potential for micro trauma
at the ends of the bones. This
micro trauma results in a section at the end of the bone not getting the blood
supply that it needs to undergo this cartilage to bone transformation. The
key here is blood supply. Something is compromising the blood to these tissues
and for years we thought it was the direct trauma of weight bearing. We
know that these animals always have subluxation reads at the caudal cervical
areas of C-6-7-8. The effects of subluxation are vaso-constriction
(impeded blood supply) and accumulation of the by-products of subluxation that
are chemical that will further degrade the developing tissues and cause pain.
Those by-products are: Lactic Acid H+ ions Seritonin Bradykinins Prostaglandins P (pain) Substance Others Whether
these substances induce the problem with ossification, or they cause the
miss-ossification and the resultant OCD, is not known. Whether this event causes
the subluxation reads or is caused by this event is also not known. In
any event, these animals are easily and definitively diagnosed with the VOM
Diagnostic Passes; long before radiographic bone changes are evident, allowing
early subluxation reduction and possibly avoidance of this condition. In
Danes, and other giant breeds prone to this problem, we begin routinely
screening them at 6-months of age and see them monthly until 14 months. A
"normal" Dane developing this condition will present without lameness.
However, when the area in the caudal cervical area is tested with the device,
the pup will almost drop to the floor. He will act as though he is being
instantaneously "shut off" and then, without experiencing pain, will
be normal again. Radiographic findings will not be seen for weeks to
months, if at all. This makes this early detection system the fastest and most
effective to date. Unfortunately,
radiologists and surgeons who will direct their excellent skills to treating the
bony problem once it can be isolated on x-ray, will tend to relegate this
approach to "mumbo jumbo" or "black magic".
The veterinary teaching institutions have not discovered these techniques, not
yet. We
have taken on the responsibility of breeding this disease into our Danes and the
giant breeds we love so much. We have explained their predilection to OCD and
other diseases of this nature to a "genetic destiny" mandated by our
breeding. I
feel this is far too harsh and somewhat irresponsible. To conclude that these
diseases are the result of improper genetics because we cannot understand them
is a "cop-out" and not worthy of these magnificent animals. Conclusion We
know that certain breeds are predisposed to subluxation. We know that
subluxations cause these diseases and that these subluxation patterns show up
before the disease is seen. We know that when we do see the disease we always
find the same pattern of subluxations and we know that the reduction of these
patterns eliminates the disease or the progression of the disease. We
have in our hands now a means for early diagnosis and treatment. One that is
fast, easy and inexpensive. As the VOM Technology grows and is accepted in the
veterinary community, the link between subluxation and disease will become more
evident. Diseases will be diagnosed and treated in this fashion and our
beloved pets will live much happier lives. They can become free of the
"medical disasters" that plague the lives of these magnificent
creatures and, of course their owners. Epilogue Wm.
L. Inman BS, DVM, CVCP is the founder and developer of the International
Association of Chiropractitioners (IAVPC). The IAVPC is dedicated to eradicating
disease in the domestic animals and uses exclusively the patented VOM Technology
that Dr. Inman discovered and developed in 1982. Based
on over 45,000 clinical cases, the VOM Technology (through VOM Seminars Inc.)
has trained over 3800 practitioners in the US and has become the most common
means of subluxation diagnosis and treatment in the world
Bibliography Akeson WII, Annie P., Savio LY: Immobility Effects on Synovial Joints, the Path mechanics of Joint Contracture, The 3rd International Congress on Biotheology:95-110, 1980. Chrisman CL: Problems in Small Animal Neurology- 1982 Dishman R: Review of the literature Supp. A Scientific Basis for the Chiropractic Subluxation Complex. JMPT 8(31): 163-174. Denslow JS: An Analysis of the Variability of Spinal Reflex Thresholds. Journal of Neurophysiology 1: 207-215 Duell ML: The Force of the Activator Adjusting Instrument. Chiro Econ. Nov/Dec 1984. Fuhr AW, Smith DW: Accuracy of Piezoelectric Accelerometers in Measuring the Distance of a Spinal Adjusting Injury Instrument. J.M.P.T. 9(1) 15-21, 1986. Giteiman R, Fitz-Ritson D: Somatovisceral Reflexes. ACA Journal 8(4) 640-671, 1984. Haldma S: The Pathophysiology of the Spinal Subject. Journal of CCA: 5-11 Sept 1975. Hall MC: Cartilage Changes after Experimental Immobilization of the Knee Joint of the Young Rat, Journal of Bone and Joint Surgery 35A(1) 36-44, 1963. Kirk RW: Current Veterinary Therapy-Small Animal Practice. 739-836, 1986. Kirkaldy-Willis: The Relationship of Structural Pathology to the Nerve Root. Spine, 9(1): 49-52. 1984. Oliver JE, Lorenz MD: Handbook of Veterinary Neurology, Saunders Co. Second Edition, 1993. Slosberg M: Understanding the Role of Diagnosis Within the Scope of Chiropractic. Chiro. Econ. Jan/Feb 70-75 1984. Vannerson JF, Nimmo RL:Specificity ans the Law of Facilitation in the Nervous System. The Receptors 21(1): 1-8, 1971. Wyke BD:Articular Neurology and Manipulative Therapy. Glasgow ed. Aspects of Manipulative Therapy, 72-77. 1988. |
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